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The rate of fall of the glycogen was linear throughout the interval of aglycemia. This advised that if glycogen was unregulated previous to introducing aglycemia then the latency to CAP failure could possibly be extended if glycogen was me Exposing the optic nerve to aglycemia after increasing glycogen content did indeed lead to a rise in the latency to CAP failure. Incubating nerves in growing concentrations of glucose such that a variety of glycogen levels have been attained resulted in a linear relationship whereby the latency to CAP failure was determined by the glycogen content i.e. growing glycogen content material within the nerve on the |
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| Alysa |
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The rate of fall of the glycogen was linear throughout the interval of aglycemia. This advised that if glycogen was unregulated previous to introducing aglycemia then the latency to CAP failure could possibly be extended if glycogen was me Exposing the optic nerve to aglycemia after increasing glycogen content did indeed lead to a rise in the latency to CAP failure. Incubating nerves in growing concentrations of glucose such that a variety of glycogen levels have been attained resulted in a linear relationship whereby the latency to CAP failure was determined by the glycogen content i.e. growing glycogen content material within the nerve on the |
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| Alysa |
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The rate of fall of the glycogen was linear throughout the interval of aglycemia. This advised that if glycogen was unregulated previous to introducing aglycemia then the latency to CAP failure could possibly be extended if glycogen was me Exposing the optic nerve to aglycemia after increasing glycogen content did indeed lead to a rise in the latency to CAP failure. Incubating nerves in growing concentrations of glucose such that a variety of glycogen levels have been attained resulted in a linear relationship whereby the latency to CAP failure was determined by the glycogen content i.e. growing glycogen content material within the nerve on the |
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